In every cell of the body lives a small but powerful guardian called p53, a protein that constantly checks our DNA for damage. When it finds a problem, p53 has a choice to make: Try to repair the damage, or If the damage is too severe, tell the cell to self-destruct before it causes harm. In healthy tissue, this process keeps us safe. But in certain diseases, that same protective system can become overactive, and that may be what happens in Amyotrophic Lateral Sclerosis (ALS)

Neurodegenerative diseases, including Motor Neurone Disease (MND)/Amyotrophic Lateral Sclerosis (ALS), Parkinson’s Disease, Huntington’s Disease, and Dementia — are often described as progressive and irreversible. But new thinking is emerging. A growing number of researchers and clinicians believe that by understanding the systems biology  of the brain, the complex network of pathways that influence energy, inflammation, and repair — we may be able to slow, and in some cases

When researchers recently mapped deaths from Amyotrophic Lateral Sclerosis (ALS) across the United States, they noticed something unexpected. The distribution wasn’t random. Certain regions consistently showed higher rates of the disease. Curious, they did the same for Multiple Sclerosis (MS) — another serious neurological condition that, like ALS, affects the brain and spinal cord. To their surprise, the two maps almost perfectly overlapped. This observation became the found